PDF) Overexpression of the LexA-regulated tisAB RNA in E. coli inhibits SOS functions; implications for regulation of the SOS response
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Non-canonical LexA proteins regulate the SOS response in the Bacteroidetes
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Mycobacterial Mutagenesis and Drug Resistance Are Controlled by Phosphorylation- and Cardiolipin-Mediated Inhibition of the RecA
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Non-canonical LexA proteins regulate the SOS response in the Bacteroidetes
Non-canonical LexA proteins regulate the SOS response in the Bacteroidetes
Non-canonical LexA proteins regulate the SOS response in the Bacteroidetes
Mycobacterial Mutagenesis and Drug Resistance Are Controlled by Phosphorylation- and Cardiolipin-Mediated Inhibition of the RecA
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Study of O-Phosphorylation Sites in Proteins Involved in Photosynthesis-Related Processes in Synechocystis sp. Strain PCC 6803: Application of the SRM Approach | Journal of Proteome Research
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Antibiotic-induced DNA damage results in a controlled loss of pH homeostasis and genome instability
Analysis of Escherichia coli RecA Interactions with LexA, CI, and UmuD by Site-Directed Mutagenesis of recA
PDF) Overexpression of the LexA-regulated tisAB RNA in E. coli inhibits SOS functions; implications for regulation of the SOS response
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Spatial and temporal organization of RecA in the Escherichia coli DNA-damage response
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Single-molecule imaging of LexA degradation in Escherichia coli elucidates regulatory mechanisms and heterogeneity of the SOS re
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Single-molecule imaging of LexA degradation in Escherichia coli elucidates regulatory mechanisms and heterogeneity of the SOS re
Mycobacterial Mutagenesis and Drug Resistance Are Controlled by Phosphorylation- and Cardiolipin-Mediated Inhibition of the RecA
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